When alcohol attaches to GABA receptors, it enhances the effects of the neurotransmitter gamma-aminobutyric acid (GABA), which is the primary inhibitory neurotransmitter in the brain. This binding increases the influx of chloride ions into neurons, leading to greater inhibition of neuronal activity. As a result, this can produce sedative, anxiolytic, and muscle-relaxing effects, contributing to the overall depressant effects of alcohol on the central nervous system.
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