The point mutation in the sex cell of the rabbit could lead to a change in the amino acid sequence of the protein responsible for building heart muscles. If this mutation alters the protein's structure or function, it could result in compromised heart muscle development or function in the offspring. However, if the mutation is silent or does not significantly affect protein function, the offspring may not exhibit any noticeable heart issues. Overall, the most likely effect would depend on the specific nature of the mutation and its impact on protein function.
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