Passive stretching of the ventricular muscle during diastole, primarily influenced by the volume of blood filling the heart, leads to an increase in myocardial fiber length. This phenomenon, known as the Frank-Starling mechanism, enhances the force of contraction during systole, allowing the heart to pump more effectively. The greater the ventricular filling (preload), the more the muscle fibers stretch, resulting in stronger contractions to accommodate the increased blood volume. This intrinsic property helps the heart maintain efficient circulation in response to varying blood return.
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